Uterus With Asherman's Syndrome

Description

Uterine cavity anatomy is the focus, with intrauterine adhesions (Asherman’s syndrome) spanning the endometrial surfaces as thick fibrous bands. From this internal perspective, the scar tissue bridges the anterior and posterior uterine walls, narrowing the central cavity from the fundus inferiorly toward the isthmus and internal cervical os. The tubal ostia at the superolateral corners can be partially obscured by adhesions that tether the cornual regions, and the normal smooth endometrial lining is interrupted where scar replaces functional mucosa. Orientation stays intraluminal, keeping attention on how opposing walls become apposed and fused. In practice, these adhesions most often follow endometrial trauma, classically postpartum curettage or repeated dilation and curettage after miscarriage, but they can also occur after infection or uterine surgery. Patients present with secondary amenorrhea or hypomenorrhea, cyclic pelvic pain from outflow obstruction, infertility, and recurrent pregnancy loss, and severe cases raise the risk of abnormal placentation (placenta accreta spectrum) once conception occurs. Hysteroscopy is the reference standard for diagnosis and treatment, and the fixed internal view here mirrors what clinicians conceptualize during hysteroscopic lysis of adhesions: where bands attach, how they partition the cavity, and which regions remain patent. Small details matter. Use this illustration in reproductive endocrinology and infertility teaching, gynecology board review, and patient-facing counseling materials that explain why a previously normal cavity can become partially obliterated after uterine instrumentation. It also supports journal figures and slide decks discussing hysteroscopic classification of adhesions, postoperative adhesion-prevention strategies, and differential diagnosis of amenorrhea with infertility. Anatomical accuracy verified by SciePro's Medical Advisory Board.